Nutmeg and Maois: Can You Take Them Together?

High — Consult Your Doctorcontraindication
Evidence-gradedLast reviewed June 1, 2026Source: PubMed (Truitt EB et al., Proc Soc Exp Biol Med 1963)
Learn about each ingredient:NutmegMaois

Quick answer

Nutmeg contains myristicin, which has weak monoamine oxidase inhibitor (MAOI) activity. Combining nutmeg supplements or large culinary doses with prescription MAOIs can theoretically precipitate hypertensive crisis or serotonin syndrome through additive MAO inhibition.

Avoid nutmeg supplements and large therapeutic doses of nutmeg essential oil if you take an MAOI such as phenelzine, tranylcypromine, isocarboxazid, or selegiline. Sprinkles of nutmeg on holiday foods are unlikely to cause problems.

What happens when you take nutmeg with MAOIs?

Nutmeg, the dried seed of Myristica fragrans, contains essential oils rich in myristicin, elemicin, and safrole. Myristicin has long been recognized as a psychoactive substance at high doses, and laboratory work dating back to the 1960s demonstrated that myristicin and whole nutmeg can inhibit monoamine oxidase (MAO), the enzyme that breaks down serotonin, norepinephrine, dopamine, tyramine, and related monoamines. The inhibition is considered weak and reversible compared to prescription MAOIs, but it is real.

MAOIs prescribed for depression (phenelzine, tranylcypromine, isocarboxazid), Parkinson's disease (selegiline, rasagiline), or smoking cessation (the antidepressant moclobemide, in countries where it is available) work by blocking MAO so that synaptic monoamines accumulate. Patients on MAOIs are warned to avoid tyramine-rich foods because tyramine, normally cleared by MAO in the gut and liver, can flood the circulation, displace norepinephrine from sympathetic nerve terminals, and trigger a hypertensive crisis with severe headache, blood pressure surge, sweating, chest pain, and the risk of intracranial hemorrhage.

When myristicin's weak MAO inhibition is layered on top of a prescription MAOI, two related risks emerge. First, the additive enzyme blockade can make the patient even more vulnerable to dietary tyramine. Second, myristicin and other nutmeg constituents may exert mild serotonergic activity, raising the theoretical risk of serotonin syndrome, a constellation of agitation, hyperreflexia, clonus, autonomic instability, and hyperthermia that can be fatal.

Why is this important?

MAOI therapy already requires considerable patient effort: aged cheeses, cured meats, fermented soy products, draft beer, and several other tyramine sources have to be avoided, and a long list of cough-cold remedies and serotonergic drugs are off-limits. Adding a herb that further inhibits MAO is exactly the wrong direction. Hypertensive crisis is a medical emergency, and serotonin syndrome can progress quickly enough that timely recognition is critical.

The risk is dose-dependent. A pinch of nutmeg in a holiday eggnog or sprinkled on a latte is unlikely to deliver enough myristicin to matter. Large doses, on the order of one tablespoon or more of ground nutmeg, are also independently dangerous because of nutmeg's own neurotoxic and anticholinergic effects (the so-called 'nutmeg high' is rarely pleasant and can cause tachycardia, hallucinations, severe nausea, and prolonged anxiety). Nutmeg essential oil used internally or in concentrated tincture form carries the highest concern.

What should you do?

If you take a prescription MAOI, treat nutmeg like any other restricted substance: trace amounts in food are generally fine, but avoid concentrated nutmeg supplements, nutmeg essential oil taken internally, and large therapeutic doses. The same caution applies to mace, which is the outer covering of the nutmeg seed and shares its essential oil profile. Watch for the warning signs of hypertensive crisis (severe occipital or temporal headache, palpitations, blood pressure spike, nausea, sweating, photophobia) and serotonin syndrome (restlessness, tremor, muscle twitching, fever, confusion). Both warrant emergency care.

If you are also taking serotonergic medications alongside an MAOI (which is itself usually contraindicated), the combined risk with nutmeg climbs even higher. SSRIs, SNRIs, tramadol, dextromethorphan, meperidine, triptans, lithium, St. John's wort, and 5-HTP should all be avoided with MAOI therapy, and nutmeg simply adds another small risk to a list that is already long.

Which specific products are affected?

Concerning sources of myristicin include: whole nutmeg or freshly ground nutmeg in large amounts; mace; nutmeg essential oil (both internal use and high-dose aromatherapy); concentrated nutmeg supplements marketed for sleep, anxiety, or 'natural mood support'; some commercial herbal sleep blends; and certain ethnobotanical preparations. Prescription MAOIs include phenelzine (Nardil), tranylcypromine (Parnate), isocarboxazid (Marplan), selegiline (Eldepryl, Emsam transdermal patch), rasagiline (Azilect), and in non-US markets, moclobemide (Aurorix, Manerix).

The 'cheese effect' washout period after stopping an irreversible MAOI is typically 14 days, during which MAO must regenerate. Nutmeg restrictions should be maintained throughout that interval and not relaxed simply because the daily pill has stopped.

The bottom line

Culinary nutmeg in normal amounts is unlikely to interact dangerously with MAOIs, but concentrated nutmeg supplements, essential oil, and large doses can add to MAOI activity and tip patients into hypertensive crisis or serotonin syndrome. If you take an MAOI, avoid nutmeg supplements entirely and keep dietary nutmeg to the levels you would find in a typical recipe.

References

Primary evidence for this article. Always consult your healthcare provider for personal medical advice.

Related Interactions

Other interactions you should know about

Yerba Mate + Maois

high

Yerba mate contains caffeine plus trace monoamine oxidase inhibitor-like compounds, but its bigger risk with prescription MAOIs (phenelzine, tranylcypromine, isocarboxazid, selegiline) is its sympathomimetic load: caffeine, theobromine, and modest tyramine content can amplify the pressor response in patients with inhibited MAO. Combining the two can trigger hypertensive crisis or serotonergic adverse effects.

Maoi + Tyramine Foods

critical

Monoamine oxidase inhibitors block MAO-A in the gut and liver, preventing the breakdown of dietary tyramine. Unmetabolized tyramine triggers a massive release of stored norepinephrine, producing a hypertensive crisis ('cheese reaction') with severe blood pressure spikes, stroke, or death.

Maoi + 5-Htp

critical

5-HTP is the direct precursor to serotonin and bypasses the rate-limiting step of serotonin synthesis. Combined with an MAOI, which blocks serotonin breakdown, intracellular and synaptic serotonin can rise to toxic levels, producing serotonin syndrome.

Maoi + St. John's Wort

critical

St. John's Wort inhibits serotonin, dopamine, and norepinephrine reuptake and has weak MAOI activity in vitro. Combined with a prescription MAOI, monoamine clearance is blocked at multiple levels, producing serotonin syndrome and/or hypertensive crisis.

Fermented Foods + Maois

critical

Fermented foods accumulate tyramine through microbial decarboxylation of tyrosine during fermentation, and MAOIs block the monoamine oxidase enzyme that normally clears tyramine from the gut and bloodstream. Unmetabolized tyramine then triggers massive norepinephrine release, which can produce a hypertensive crisis with systolic blood pressure spiking above 180 mmHg.

Cayenne + Ace Inhibitors

low

Capsaicin, the active component of cayenne, can trigger or worsen the dry cough characteristic of ACE inhibitor therapy by sensitizing airway cough receptors. A published case report describes topical capsaicin inducing cough in a patient already on an ACE inhibitor.

Disclaimer: This article is for informational purposes only and is not a substitute for professional medical advice. Always consult your healthcare provider before making changes to your supplement or medication routine. Pilora does not diagnose, treat, cure, or prevent any disease.

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