Urolithin A

phytochemicalpolyphenol metabolite
Best with a meal

At a glance

Best for
middle-aged and older adults targeting muscle mitochondrial health and endurance
Typical dose
500–1000 mg/day with food
Time to effect
Weeks to a few months
Main caution
Limited long-term safety data; avoid in pregnancy and breastfeeding
Evidence strength: Good for mitochondrial/endurance markers; modest and inconsistent for raw strength

What is it

Urolithin A is a metabolite produced by gut bacteria from ellagitannins and ellagic acid, polyphenols found in pomegranates, walnuts, and certain berries. The body cannot make urolithin A directly from these foods; instead the right gut microbes convert the dietary precursors into it. Because only a portion of people harbour the bacteria needed to produce meaningful amounts, urolithin A is increasingly sold as a supplement so that the active compound can be delivered directly regardless of gut flora.

Is it worth it for you?

Worth considering if…

  • You are middle-aged or older and want to support muscle mitochondrial function
  • You will take it consistently for one to several months
  • You do not reliably produce urolithin A from food (most people)

Probably skip if…

  • You expect large gains in raw strength or athletic performance
  • You want an acute pre-workout effect
  • You are pregnant or breastfeeding

Evidence at a glance

GoalEvidenceEffectBest fitTime
muscle endurance and mitochondrial healthLimitedModest improvements in muscle endurance and mitochondrial markersmiddle-aged and older adults with declining muscle functionWeeks to months
cellular and mitochondrial ageingLimitedImproved mitophagy and mitochondrial biomarkersadults interested in mitochondrial-ageing pathwaysWeeks
exercise capacity in healthy adultsLimitedInconsistent; small if anyactive adults hoping to extend exercise capacityWeeks to months

Evidence for 3 uses

AI-assisted evidence assessment — talk to your doctor before relying on any single supplement.

muscle endurance and mitochondrial health

Supplement benefit
Limited

Urolithin A stimulates mitophagy, the clearance of damaged mitochondria, and randomized trials in middle-aged and older adults report improved muscle mitochondrial gene expression, mitochondrial-health markers, and some measures of muscle endurance. Effects on raw strength and peak performance are more modest and not always significant. Because only a minority make it efficiently from food, direct supplementation delivers a consistent dose.

Effect size: Modest improvements in muscle endurance and mitochondrial markers
Time to effect: Weeks to months
Best fit: middle-aged and older adults with declining muscle function
Less likely: young, well-trained athletes seeking strength gains

Bottom line: Urolithin A modestly improves muscle endurance and mitochondrial markers in older adults, with smaller effects on strength.

cellular and mitochondrial ageing

Mechanism only
Limited

In worms and aged mice, urolithin A extends lifespan and improves muscle function, and human studies show changes in mitochondrial gene expression and biomarkers. Evidence for slowing human ageing rests on preclinical models and biomarker changes, not clinical ageing outcomes. This claim is mechanistic and should not be read as proven anti-ageing benefit.

Effect size: Improved mitophagy and mitochondrial biomarkers
Time to effect: Weeks
Best fit: adults interested in mitochondrial-ageing pathways

Bottom line: Anti-ageing claims rest on animal and biomarker data, not human clinical outcomes.

exercise capacity in healthy adults

Supplement benefit
Limited

Trials examining whole-body exercise performance and aerobic capacity in healthy adults have shown inconsistent results, with mitochondrial and endurance markers improving more reliably than overall performance. Confidence for a meaningful exercise-capacity benefit is low. It is better framed as muscle-quality support than a performance enhancer.

Effect size: Inconsistent; small if any
Time to effect: Weeks to months
Best fit: active adults hoping to extend exercise capacity
Less likely: trained athletes expecting clear performance gains

Bottom line: Urolithin A does not reliably boost overall exercise capacity in healthy adults.

Evidence is mixed

Mitochondrial and endurance markers improve, but effects on whole-body exercise performance are inconsistent across trials.

How it works

Urolithin A's most studied action is the stimulation of mitophagy, the cellular housekeeping process that identifies and recycles damaged or dysfunctional mitochondria. Mitochondrial quality declines with age, particularly in energy-hungry tissues like skeletal muscle, and impaired mitophagy is thought to contribute to the loss of muscle strength and endurance over time. By promoting the clearance of worn-out mitochondria and encouraging the growth of healthy new ones, urolithin A is proposed to improve cellular energy production and resilience. In preclinical models, urolithin A extends lifespan in worms and improves running endurance and muscle function in aged mice. In humans, randomised trials in middle-aged and older adults have reported improvements in muscle mitochondrial gene expression, markers of mitochondrial health, and measures of muscle endurance, although effects on raw strength and exercise performance have been more modest and not always significant. Only a minority of people produce urolithin A efficiently from food, which is the central rationale for direct supplementation.

How to take it

Typical dose
500–1000 mg/day (250 mg also studied)
Timing
Once daily with a meal
With food
Take with food, ideally containing some fat
How long to try
One to four months of consistent use

What to track

  • muscle endurance and fatigue
  • exercise capacity
  • consistency over weeks

2 commercial forms

Synthetic urolithin A (defined ingredient)

Delivers a consistent dose directly, bypassing the need for gut bacteria to produce it from dietary precursors.

A standardised, highly pure form produced by synthesis rather than extraction; this is the form used in most clinical trials.

Pomegranate extract (ellagitannin source)

Conversion to urolithin A depends on the individual's gut bacteria; only a minority of people produce substantial amounts.

Provides the dietary precursors (ellagic acid and ellagitannins) rather than urolithin A itself.

Safety

Common side effects

mild, transient digestive complaints (no more than placebo in trials)

Who should avoid it

  • pregnant and breastfeeding women
  • people on multiple medications or scheduled for surgery without clinician input

Pregnancy & breastfeeding

Insufficient safety data; avoid in pregnancy and breastfeeding.

Interactions

No clinically significant drug interactions have been established for urolithin A. As with many polyphenol-derived compounds, theoretical effects on drug-metabolising enzymes cannot be fully excluded, so people taking medications with a narrow therapeutic window should mention any supplement use to their clinician. There is no strong evidence that urolithin A meaningfully alters the metabolism of common medications.

Food sources

FoodAmount%DV
Pomegranate (juice and arils)provides ellagitannin precursors, not urolithin A directly
Walnutsrich in ellagitannins (precursors)
Raspberriescontain ellagic acid (precursor)
Strawberriescontain ellagic acid (precursor)

Choosing a product

Look for

  • defined urolithin A ingredient (e.g. GRAS-notified)
  • stated dose 250–1000 mg
  • third-party tested

Be skeptical of

  • reverses ageing
  • guaranteed strength or performance gains
  • longevity cure claims

Frequently asked questions

Can I just eat pomegranates instead of supplementing?

Sometimes. Pomegranates and walnuts provide the precursors, but your gut bacteria have to convert them into urolithin A. Studies suggest only around a third to a half of people produce meaningful amounts, which is why a direct supplement gives a more reliable dose.

What is mitophagy and why does it matter?

Mitophagy is the cell's process for recycling damaged mitochondria, the structures that produce energy. It declines with age. Urolithin A's main proposed benefit is stimulating mitophagy to keep muscle and other tissues energetically healthy.

Will urolithin A make me stronger?

The clearest human evidence is for improved muscle endurance and mitochondrial markers, not raw strength. Effects on maximal strength and performance have been modest and inconsistent across trials.

What dose is used in studies?

Most clinical trials used 500 mg or 1000 mg per day with food for one to four months. Lower doses have also been tested.

Is urolithin A safe?

In trials lasting up to a few months at doses up to 1000 mg per day it was well tolerated, with side effects similar to placebo. Long-term safety over years is not yet established.

References by claim

muscle endurance and mitochondrial health

  • Singh et al., 2022PMC (2022) link
  • Liu et al., 2022PMC (2022) link

cellular and mitochondrial ageing

  • Denk et al., 2025PMC (2025) link
  • Denk et al., 2026PMC (2026) link

exercise capacity in healthy adults

  • Zhao et al., 2024PMC (2024) link

Track Urolithin A with Pilora

Set up dose reminders, check interactions, and join the community in the Pilora iPhone app.

Coming to App Store
Evidence-based·Last reviewed May 30, 2026·Evidence current as of May 30, 2026·How we grade evidence

Disclaimer: These statements have not been evaluated by the FDA. This page is educational, not a substitute for personalized medical advice. Evidence grades are AI-assisted assessments — talk to your doctor before starting any new supplement, especially if you’re pregnant, breastfeeding, on medications, or managing a chronic condition.