Chrysin

phytochemicalflavonoid
Best with a meal

At a glance

Best for
No well-established clinical use in humans at this time
Typical dose
500–1,500 mg/day (common supplement range, poorly absorbed)
Time to effect
Unknown — no demonstrated clinical effect in humans
Main caution
Oral bioavailability is under 1% — aromatase inhibition demonstrated in vitro does not translate to measurable hormone changes in humans
Evidence strength: Evidence for marketed benefits (testosterone support, hormone balance) is essentially absent in controlled human trials

What is it

Chrysin (5,7-dihydroxyflavone) is a flavone found in passionflower, propolis, and honey. It is marketed as a natural aromatase inhibitor and testosterone booster, despite weak human evidence.

Is it worth it for you?

Worth considering if…

  • You are exploring it as a low-risk flavonoid antioxidant without expectation of hormone effects

Probably skip if…

  • You are expecting testosterone elevation or estrogen reduction — human trials show no measurable effect
  • You have a hormone-sensitive condition (discuss with clinician before any 'aromatase inhibitor')
  • You are pregnant or breastfeeding

Evidence at a glance

GoalEvidenceEffectBest fitTime
antioxidant activityMixedAntioxidant effects in cell-based models; not established in human tissue at typical oral dosesNot established for any human populationUnknown in human context
testosterone support via aromatase inhibitionMixedNo measurable testosterone or estrogen change in controlled human trialsNo population for which benefit has been demonstratedNot established

Evidence for 2 uses

AI-assisted evidence assessment — talk to your doctor before relying on any single supplement.

antioxidant activity

Mechanism only
Mixed

Chrysin exhibits antioxidant and anti-inflammatory activity in cell culture and animal models. However, oral bioavailability is under 1% due to extensive first-pass glucuronidation, making it unlikely that meaningful concentrations reach target tissues after oral supplementation in humans.

Effect size: Antioxidant effects in cell-based models; not established in human tissue at typical oral doses
Time to effect: Unknown in human context
Best fit: Not established for any human population

Bottom line: Antioxidant effects are a cell-culture phenomenon with no meaningful human clinical evidence at achievable oral doses.

testosterone support via aromatase inhibition

Mechanism only
Mixed

Chrysin inhibits aromatase in vitro at high concentrations. In controlled human trials, oral chrysin supplementation (5001,000 mg/day) has not produced measurable changes in serum testosterone, estradiol, or luteinizing hormone. The low bioavailability of oral chrysin (<1%) is the primary reason in vitro findings do not translate to human hormone effects.

Effect size: No measurable testosterone or estrogen change in controlled human trials
Time to effect: Not established
Best fit: No population for which benefit has been demonstrated

Bottom line: Does not raise testosterone or meaningfully inhibit aromatase in humans at supplement doses — the marketed mechanism does not work orally.

Evidence is mixed

In vitro aromatase inhibition is well documented, but all controlled human trials show no measurable hormonal effect at typical oral doses, consistent with the known near-zero bioavailability.

How it works

Chrysin inhibits aromatase (the enzyme converting testosterone to estrogen) in vitro. In humans, oral chrysin has very poor bioavailability (under 1%) due to extensive first-pass glucuronidation, so the in-vitro aromatase inhibition does not translate to measurable hormone changes at typical doses.

How to take it

Typical dose
500–1,500 mg/day if trialing
Timing
With meals to reduce GI upset
With food
With food
How long to try
No standard trial period established; effects in humans have not been demonstrated

What to track

  • Total testosterone and estradiol if trialing for hormone effects (expect no change based on current evidence)
  • Any GI discomfort

2 commercial forms

Standard chrysin

Very low oral bioavailability.

Standard capsule form.

Chrysin + piperine

Piperine claimed to improve absorption.

Marketing exceeds evidence.

Safety

Common side effects

Mild GI upset

Who should avoid it

  • Pregnant and breastfeeding women (insufficient data, hormonal claims)
  • People with hormone-sensitive conditions without clinician guidance

Pregnancy & breastfeeding

Insufficient safety data; avoid during pregnancy and breastfeeding.

Interactions

CYP enzyme substratesMinor

May inhibit certain CYP enzymes and UGTs at high doses; practical impact at typical doses unclear

Food sources

FoodAmount%DV
Bee propolistrace flavone content
Honey (1 tbsp)trace chrysin

Choosing a product

Look for

  • Chrysin purity and source specified
  • Dose per serving clearly stated

Be skeptical of

  • 'Natural testosterone booster'
  • 'Aromatase inhibitor'
  • 'Raises free testosterone'
  • 'Lowers estrogen naturally'

Frequently asked questions

Does chrysin boost testosterone?

Human evidence does not support this claim; oral chrysin has very poor absorption.

Is chrysin safe?

Short-term safety appears acceptable, but long-term and hormonal effects are not well studied.

References by claim

antioxidant activity

  • Naz et al., 2019PubMed (2019) link
  • Talebi et al., 2021PubMed (2021) link

Track Chrysin with Pilora

Set up dose reminders, check interactions, and join the community in the Pilora iPhone app.

Coming to App Store
Evidence-based·Last reviewed May 30, 2026·Evidence current as of May 30, 2026·How we grade evidence

Disclaimer: These statements have not been evaluated by the FDA. This page is educational, not a substitute for personalized medical advice. Evidence grades are AI-assisted assessments — talk to your doctor before starting any new supplement, especially if you’re pregnant, breastfeeding, on medications, or managing a chronic condition.