enzyme induction

8 interactions related to enzyme induction

verapamil + st. john's wort

St. John's wort is a potent inducer of intestinal CYP3A4 and P-glycoprotein. In a controlled study, two weeks of St. John's wort reduced the AUC of R- and S-verapamil by roughly 78-80%, dramatically lowering systemic drug exposure and likely therapeutic effect.

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verapamilst johns worthypericum perforatumcyp3a4p-glycoproteincalcium channel blockerherbal interactionenzyme induction

digoxin + st. john's wort

St. John's wort induces intestinal P-glycoprotein, increasing efflux of digoxin and reducing its absorption. Controlled studies show digoxin AUC falls roughly 25% and peak concentrations around 30-36% after two weeks of St. John's wort, potentially producing therapeutic failure in rate control or heart failure management.

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digoxinst johns worthypericum perforatump-glycoproteincyp3a4cardiac glycosideherbal interactionenzyme induction

phenytoin + st. john's wort

St. John's Wort is a potent inducer of CYP3A4, CYP2C9, CYP2C19, and P-glycoprotein via activation of the pregnane X receptor. Because phenytoin is heavily metabolized by CYP2C9 and CYP2C19, concurrent St. John's Wort can lower phenytoin plasma concentrations into the subtherapeutic range, increasing the risk of breakthrough seizures.

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phenytoindilantinst johns worthypericumcyp2c9cyp2c19enzyme inductionepilepsyseizure breakthrough

phenytoin + ginkgo

Ginkgo biloba induces CYP2C19, the primary enzyme responsible for phenytoin metabolism. A published case report described a fatal breakthrough seizure in a patient on phenytoin and valproate whose autopsy revealed subtherapeutic anticonvulsant levels and self-administration of ginkgo biloba. Ginkgo also has independent pro-convulsant potential at high doses and through ginkgotoxin contamination.

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phenytoindilantinginkgo bilobacyp2c19enzyme inductionepilepsyseizure breakthroughginkgotoxin

oxycodone + st. john's wort

St. John's Wort strongly induces CYP3A4, the main enzyme that metabolizes oxycodone. In a controlled crossover trial, St. John's Wort cut oral oxycodone plasma exposure (AUC) by roughly 50% and significantly reduced its analgesic effect.

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oxycodonest johns wortcyp3a4opioidpain managementherbal interactionenzyme inductionanalgesia

phenytoin + folate

Phenytoin lowers serum and red-cell folate through enzyme induction and impaired absorption of polyglutamate folates, but high-dose folate supplementation in turn accelerates phenytoin metabolism and can drop drug levels enough to cause seizure breakthrough.

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phenytoindilantinfolatefolic acidanticonvulsantepilepsyseizuresenzyme inductiondeficiency

omeprazole + st. john's wort

St. John's wort potently induces CYP3A4 and CYP2C19, the enzymes responsible for omeprazole metabolism. Co-administration significantly lowers omeprazole plasma concentrations, reducing its acid-suppressing efficacy and potentially compromising treatment of GERD, ulcers, or H. pylori eradication.

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omeprazolest johns worthypericumcyp3a4cyp2c19enzyme inductionppiherbal interaction

carbamazepine + st. john's wort

Both carbamazepine and St. John's Wort are strong inducers of CYP3A4, the enzyme that primarily metabolizes carbamazepine. Although healthy-volunteer studies have shown limited additional effect on chronic carbamazepine kinetics (because carbamazepine already maximally autoinduces its own metabolism), starting or stopping St. John's Wort can destabilize carbamazepine levels, and the herb can lower exposure to single carbamazepine doses by up to 21% before autoinduction is established.

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carbamazepinetegretolst johns worthypericumcyp3a4enzyme inductionautoinductionepilepsy